An enantiomer of ibuprofen that does not inhibit COX; inhibits NF-κB activation (IC50 = 121.8 µM) in response to T-cell stimulation; blocks superoxide formation, β-glucuronidase release, and LTB4 generation by stimulated neutrophils (IC50s = 40-100 µM); can be inverted to (S)-ibuprofen in humans after oral administration.